4 Legal References for SMBs and Informed Readers ? Small, Mobile ...

Whether or not you are a small business owner, you are probably exposed to legal topics on a regular basis. There are daily news developments regarding patent lawsuits, especially in the mobile arena. ?I?m not a lawyer? (INAL) is a common phrase you?ll see on message boards and blogs as individuals seek informal guidance on areas such as business formation and Intellectual Property (IP) issues.

While you always have the option of consulting a licensed attorney, you may first want to do your own research so you can have an informed conversation. I?ve found the publications from Nolo? to be extremely helpful for general and business-specific legal knowledge. Here are some recommendations from my personal library.

Nolo?s Encyclopedia of Everyday Law ? covers a variety of subjects for your business and home, and is a good first step when you begin your research.

Form Your Own Limited Liability Company ? Covers what an LLC is and compares it to other business entities. Member/manager responsibilities and tax issues are covered in detail. Takes you step by step through the process of forming your own LLC. Nolo has similar references available should you choose another entity type such as an S Corp, C Corp or Partnership.

Legal Guide to Web & Software Development ? covers areas such as licensing agreements, contracts and IP law (trademarks, copyrights, trade secrets and patents). Whether you are building your own web site or software, or contracting out development projects, this is a very useful guide.

Trademark: Legal Care for Your Business & Product Name ? from your company and product names to your logo, trademarks are something you?ll want to learn about. What exactly is the difference between TM, SM and ?, when can you use them, and what can be trademarked? You?ll find out here. You will also learn how to conduct a trademark search, which is helpful if you are marketing a new product or service. A registered trademark will provide the most protection. This guide also walks you through the process of registering your trademark with the U.S. Patent & Trademark Office.

For tablet and e-reader users, these books are available in electronic format, so you can refer to them any time no matter where you are.

Klisto? Mobile Database for the iPad?, iPhone? and iPod touch? is now available on the Apple AppStore. Your data synced on your devices and the cloud.

?Nolo? is a federally registered trademark. All trademarks appearing here are the property of their respective owners. This article is not legal advice and I am not a lawyer. If you require professional assistance you should consult with an attorney licensed in your state. I figured I should add this paragraph based on what I learned in the books I just mentioned =)

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Source: http://smallmobile.infogenium.com/2012/11/30/4-legal-references-for-smbs/

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Louisville To BCS Bowl: Teddy Bridgewater Leads Cardinals Past Rutgers, 20-17

PISCATAWAY, N.J. ? Teddy Bridgewater couldn't take a snap from center because of a broken wrist, and had a hitch in his step because of a sore ankle.

The Louisville quarterback was too banged-up to start, but more than well enough to finish off Rutgers and send the Cardinals to the Bowl Championship Series.

Bridgewater came off the bench to throw two-second half touchdown passes, and John Wallace kicked a 29-yard field goal with 1:41 left to give Louisville a 20-17 victory against Rutgers on Thursday night in what was essentially the Big East championship game.

In a game between one team headed to the Big Ten and another bound for the Atlantic Coast Conference, the Big East handed out its second-to-last BCS bid to a school that entered the league during its last massive rebuild in 2005 and watched its athletic program blossom.

The Cardinals will be going to the BCS for the second time, first since 2006. Either the Orange or Sugar Bowl will be the destination.

Louisville (10-2, 5-2) will share this Big East title with Rutgers (9-3, 5-2), Syracuse and possibly even Cincinnati, but those guys only get a banner. The Cardinals get the ticket to the big game. The BCS standings will be used to break the tie and there is no doubt Louisville, with the best overall record in the conference, will be on top.

Bridgewater didn't start a week after getting roughed up in a loss to Connecticut. He broke his left wrist and sprained his right ankle. He entered for the first time against Rutgers in the second quarter and finished 20 for 28 for 263 yards and threw a pair of TD passes in the third quarter to wipe out a 14-3 deficit.

"They have a tremendous playmaker at the quarterback position," first-year Rutgers coach Kyle Flood said. "We just weren't able to match him in the end."

Cardinals linebacker James Burgess picked off a pass that bounced off Timmy Wright's hands with 3:53 left in the fourth quarter in Rutgers territory, and Bridgewater hit Andrell Smith on a slant for 30 yards to put Louisville in field goal range. Wallace booted through the short kick to give Louisville a three-point lead.

Rutgers' last chance ended when Gary Nova threw deep, but his receiver stopped short. Terrell Floyd made an over the shoulder interception with 1:06 left. Nova bent over and grabbed his helmet in disgust.

The Scarlet Knights, the only team that has played in the Big East since it started playing football in 1991, probably will have only one more shot to win the conference for the first time before moving to the Big Ten.

Louisville can add one of the biggest wins in school history to an already memorable week for the school, which was in Conference USA just eight years ago. On Wednesday, Louisville announced it was joining the ACC after at least one more season in the Big East.

Rutgers was up 14-3 at half after Nova hooked up with his big receivers for two long TD passes ? an 85-yarder to Brandon Coleman and a 68-yard play by Mark Harrison. Nova finished 13 for 28 for 284 yards.

The Scarlet Knights were poised for another big play in the third quarter when a fake field goal turned into a touchdown pass from holder J.T. Tartacoff to DC Jefferson. It would have made the score 21-3, but an illegal man down field penalty wiped out the play and Rutgers punted from the Louisville 31.

Everything changed from there.

Bridgewater and the Cardinals put together a 90-yard drive that took 14 plays and nearly eight minutes and cut the lead to 14-10 when the quarterback flipped a pass to Jeremy Wright, who scooted 14 yards for a touchdown.

The Cardinals needed only 16 more seconds to take the lead.

Burgess stuck kick returner Jeremy Dearing in the chest, forcing a fumble recovered by Calvin Pryor. Bridgewater went right to work, floating a perfect deep ball to DeVante Parker for a 20-yard touchdown pass.

Just like that, it was 17-14 and Scarlet Knights fans fell silent.

"It ended up a 21-point swing in a very short amount of time," Flood said. "We were never able to swing the momentum back, and that's my job."

Bridgewater had the Cardinals on the move again in the fourth quarter when a high pass bounced off the hands of Eli Rogers and into the arms of Rutgers' Lorenzo Waters, who returned the interception to the Louisville 42.

The Scarlet Knights turned the turnover into Nick Borgese's tying 38-yard field goal with 7:48 left.

Fans waved white towels, and AC/DC blared from the loud speakers. But that was the last big play Rutgers could muster.

"I think we have a football team right now that's hurting," Flood said.

Louisville ended up tossing oranges in the air when it was over and exchanging high-fives with a small group of its fans in a corner of the stadium.

The Cardinals might end up in New Orleans instead of Miami, depending on how the rest of championship weekend plays out. Surely, they will be fine with either.

___

Follow Ralph D. Russo at www.Twitter.com/ralphdrussoap

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Source: http://www.huffingtonpost.com/2012/11/30/bridgewater-leads-louisvi_0_n_2215712.html

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Skeletons in cave reveal Mediterranean secrets

ScienceDaily (Nov. 28, 2012) ? Skeletal remains in an island cave in Favignana, Italy, reveal that modern humans first settled in Sicily around the time of the last ice age and despite living on Mediterranean islands, ate little seafood. The research is published November 28 in the open access journal PLOS ONE by Marcello Mannino and colleagues from the Max Planck Institute for Evolutionary Anthropology, Germany.

Genetic analysis of the bones discovered in caves on the Egadi islands provides some of the first mitochondrial DNA data available for early humans from the Mediterranean region, a crucial piece of evidence in ancestry analysis. This analysis reveals the time when modern humans reached these islands. Mannino says, "The definitive peopling of Sicily by modern humans only occurred at the peak of the last ice age, around 19,000 -26,500 years ago, when sea levels were low enough to expose a land bridge between the island and the Italian peninsula."

The authors also analyzed the chemical composition of the human remains and found that these early settlers retained their hunter-gatherer lifestyles, relying on terrestrial animals rather than marine sources for meat. According to the study, despite living on islands during a time when sea level rise was rapid enough to change within a single human lifetime, these early settlers appear to have made little use of the marine resources available to them. The authors conclude, "These findings have crucial implications for studies of the role of seafood in the diet of Mediterranean hunter-gatherers."

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Journal Reference:

1. Marcello A. Mannino, Giulio Catalano, Sahra Talamo, Giovanni Mannino, Rosaria Di Salvo, Vittoria Schimmenti, Carles Lalueza-Fox, Andrea Messina, Daria Petruso, David Caramelli, Michael P. Richards, Luca Sineo. Origin and Diet of the Prehistoric Hunter-Gatherers on the Mediterranean Island of Favignana (?gadi Islands, Sicily). PLoS ONE, 2012; 7 (11): e49802 DOI: 10.1371/journal.pone.0049802

Note: If no author is given, the source is cited instead.

Disclaimer: Views expressed in this article do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/most_popular/~3/HoDbhCcFLcw/121128182945.htm

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With Some Geeky Tweaking, China's Web Users Can Easily Leap ...

While it?s worth remembering that most Chinese web users don?t bother to circumvent the Great Firewall internet restrictions (as folks have ample homegrown social and entertainment services), the WSJ reckons that more people than ever are actually leaping the wall to check out YouTube or Twitter thanks to some fairly simple geeky tweaking.

All it takes is for the ?hosts? file inside a Windows or Mac computer ? or an Android phone ? to be modified with a list of alternative IP addresses for a blocked site you might want to access. This can be done for free. Increasingly, more and more tech-savvy Chinese web users are helping out by maintaining updated lists of which hosts tweaks still work, and then making these lists available online. Right now, these can be found all over the web in Chinese, even on Baidu?s Wenku e-books platform, as pictured here:

Crowdsourced lists of ?host? file tweaks help Chinese web users access blocked sites such as YouTube. Click to enlarge.

One such individual helping his fellow netizens leap the Great Firewall is Felix Hsu, whom the WSJ talked to. He says, ?Lots of netizens are eager to get to know what?s happening behind the wall, but it?s not easy for them.? Felix helps run a site (sadly, no link is given) that collates these workarounds. He explains that the free service they?re providing (which was started in October 2011) saw a massive spike in traffic recently as China?s leadership changeover was met with deliberate throttling and slowing of many Chinese internet services. For one 24-hour period at the start of this month, every single Google service was blocked in China ? before some of them (obviously not the long-blocked ones like G+ or YouTube) became accessible again.

I get the feeling that most Chinese people who do this are only interested in finding fun things that most of do to pass the time on the web, such as watching kitten videos on YouTube. Nonetheless, Chinese authorities keep a close eye on overseas sites and often block those that are deemed to be carrying news that?s too controversial. That?s why the New York Times is the latest high-profile site to be blocked in the country.

[Source: WSJ China Real Time blog]

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About Steven Millward

Steven follows the shininess and brilliance of gadgets, social media and other cultural phenomena across Asia. Specialist areas of research include e-commerce, Android, smartphone adoption, and apps in general. He's currently based near Shanghai. If you have any tips or feedback, contact him via email, or on his Weibo or Twitter.

Source: http://www.techinasia.com/china-leap-great-firewall-host-file/

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Last chance to win some great baby, toddler, pre-schooler and ...

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Source: http://www.smallstepsonline.co.uk/news/4306/last-chance-to-win-some-great-baby-toddler-pre-schooler-and-family-prizes-this-month

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The Wendy's College Classic: U of R Women set the tone

The Wendy's College Classic is local college basketball at its best. The classic tips off Tuesday night featuring the men's and women's teams from eight area college.

The University of Rochester women are seven-time defending champions of The Wendy's College Classic. And despite a promising start to the season last year, the Lady YellowJackets fell flat. Now is their chance to set a new tone.

It's a week in the fall Jim Scheible always looks forward to, and why not, for the past seven seasons his Lady YellowJackets have won the longest in-season college basketball tournament in the nation.

Jim Scheible, University of Rochester Women's Head Basketball Coach, said, ?Whenever you bring all the teams together its incentive to win and winning it brings the college basketball community together.?

The University of Rochester have topped The Wendy's Classic and are seven time defending champions. But the veteran coach wants his team to remember how last season ended. After starting 17-0 and ranked 5th in the nation with a sure shot at the NCAA tournament, U of R lost six of its final eight games and missed the tournament for the first time in seven years.

Jackie Walker, senior, said, ? It was hard a rough ending. We don't want to repeat. We have the motivation, we are treating this step by step and hone in on each opponent and shutting them down.?

Amy Woods, senior, said, ?Definitely motivating. We worked hard all off season. We are taking it one by one and hopefully we will be successful.?

In sports, taking a step back often leads to a giant step forward. Scheibel is counting on it, making a statement this week is a good start, but his team is eyeing the postseason prize and he doesn't want them to lose sight of it.

Scheibel said, ?I don't think we took it for granted and you have to keep playing if you play you keep playing like it's the first time for all these players.?

Women's Schedule:
Tuesday:

U of R vs RIT at 6:00p.m. at William Smith College
Nazareth vs. SUNY Geneseo at 6:00p.m. at St. John Fisher College
Roberts Wesleyan vs. William Smith at 8:00p.m. at William Smith College
Brockport vs. St. John Fisher at 8:00p.m. at St. John Fisher College

Men's Schedule
Wednesday:

Nazareth vs. SUNY Geneseo at 6:00p.m. at St. John Fisher College
RIT vs. U of R at 6:00p.m. at Hobart College
Brockport vs. St. John Fisher at St. John Fisher College at 8:00p.m.
Roberts Wesleyan vs. Hobart at Hobart College at 8:00p.m.
?

Source: http://brighton-pittsford.whec.com/news/news/88215-wendys-college-classic-u-r-women-set-tone

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Nanotech 'second skin' could protect soldiers from chemical threats

5 hrs.

Devin Coldewey , NBC News

Advances in nanotech have allowed research into materials that actively detect and shut out harmful molecules, or even shed layers like real skin. It?could be a lifesaver for soldiers and emergency workers.

A soldier in the field has lots of protection: A helmet, flak jacket, armored panels and so on. But when it comes to chemical and biological warfare, that equipment is virtually useless. Gases and bacteria can penetrate or simply cling to soldier's garments and gear, wreaking havoc later despite being highly diffuse.

The risk can't be mitigated entirely, but polymer scientists at the University of Massachusetts at Amherst and Lawrence Livermore National Laboratory are working on a "second skin" made of?smart materials that would be a far more effective and sophisticated defense than we have now.

They call the fabric they're working on a "hybrid functional material," and it's made of a combination of carbon nanotubes and specially?designed nanoparticles. It's designed not just to block out unwanted agents, like poisonous gas molecules and spores, but to be normally breathable as well in less dangerous situations.

At a molecular level, the nanotubes are aligned vertically and arranged to form pores just a few nanometers wide. While ordinary air can pass through these tiny "nanopores," larger molecules and organisms like viruses simply don't fit.

There are smaller threats, however: Nerve agents and mustard gas would go right through even such tiny gaps. The nanomaterial?membrane?would be coated with "responsive functional groups," special molecules designed to respond to the presence of such deadly toxins. Activated molecules would trigger the nanopores to shut completely.

Lastly, in the case of agents that tend to stick around instead of diffusing, such as anthrax, the material will be designed to slough them?off, mimicking human skin. That way a garment exposed to infection wouldn't bring it back to, or out of, a hospital or base.

The leader of the research team is LLNL's Franceso Fornasiero, who is working closely with UMA's Kenneth Carter and James Watkins and others. They've just received a $1.8 million five-year grant to research how to manufacture these nanomaterials in bulk, and they estimate that new nano-uniforms could be in the field within 10 years.

Devin Coldewey is a contributing writer for NBC?News Digital. His personal website is?coldewey.cc.

Source: http://www.nbcnews.com/technology/futureoftech/nanotech-second-skin-could-protect-soldiers-chemical-threats-1C7291458

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Small businesses to Congress: Tax reforms, not tax hikes | The Daily ...

A coalition of the nation?s leading small businesses wrote a letter to Congress Tuesday asking that they enact tax reform over raising tax rates.

?[W]e strongly urge Congress to pursue comprehensive tax reform that lowers rates on all forms of business income while enacting significant entitlement reforms that put the federal budget on a sustainable fiscal path,? the letter pleas.

The letter, addressed to Senate Majority Leader Harry Reid, Senate Minority Leader Mitch McConnell, House Speaker John Boehner and House Minority Leader Nancy Pelosi, discusses the issues of both the immediate fiscal cliff problem, as well as the debt ceiling, which will once again be reached in early 2013.

?Congress should seek to enact comprehensive tax reform that simplifies the tax code and?encourages economic growth for both pass-through businesses and corporations,? the letter advises.

President Obama has called for a balanced approach to handle the automatic tax increases and budget cuts scheduling commence at the start of 2013, asking the Republicans to give on revenues.

?The reality is closing loopholes and ending deductions as an alternative to raising rates on the top earners, the top 2 percent ? sounds good, but you have to look at the context of the actual proposals,? White?House?Spokesman Jay Carney told reporters at a briefing Tuesday.

Senate Majority Leader Harry Reid said Tuesday that he supports the president?s proposal to address the debt ceiling at the same time as the fiscal cliff.

?We would be somewhat foolish to work out something on stopping us from going over the cliff and then a month or six weeks later, Republicans pull the same game they did before and say, ?We?re not going to do anything unless this happens, or we?re not going to agree to increase the debt ceiling,?? he said at a news conference.

The letter addresses the negative impacts the fiscal uncertainty is already having on the economy.

?[T]wo-thirds of business owners cite the?uncertainty over future fiscal policy as making it more difficult for them to grow their businesses and?increase employment. At the same time, the rate of business creation is at its lowest level in two?decades.?

The coalition calls on Congress to enact comprehensive tax reform as opposed to simply raising marginal tax rates on employers. They emphasize the importance of tax simplification that will encourage economic growth.

The letter is signed by 42 business groups that claim to represent ?millions of pass-through businesses employing tens of millions of?workers.?

?[W]e are eager to see Congress enact permanent, comprehensive tax reform, but this alone will?not solve the long-term fiscal imbalance,? the letter concludes.

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Source: http://dailycaller.com/2012/11/28/small-businesses-to-congress-tax-reforms-not-tax-hikes/

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How To Unlock All Sonic & All-Stars Racing Transformed Characters

How to unlock all secret Sonic & All-Stars Racing Transformed characters? That question will be answered in this guide that shows you how and who to unlock to get the full roster.

Although less than half of the initial roster is available from the get-go. Once you get all unlockable characters ? 23 out of 25 on Xbox 360 for example, but it depends on your version ? then you?ll see the Sonic & All-Stars Racing Transformed characters select screen has room for bonus characters.

Sega has hinted more than the current playable characters could be made available in Sonic & All-Stars Racing Transformed through downloadable content after release, since that?s how Metal Sonic gets unlocked (details below). The first of these would-be DLC characters is shrouded in mystery, but they would add to the racing game?s total of 26 characters across the different versions.

Table of Contents

Below we?ve listed the secret characters in alphabetical order. Take the following steps to unlock each one.

• AGES (Dreamcast VMU; new in Sonic & All-Stars Racing Transformed)

  How To Unlock: In World Tour Mode: Superstar Showdown win Ranger Rush (Sprint). Then you can buy it for 165 Stars.
  Tip: To help you win races, try and get a level 3 boost at the starting line every time, during the countdown. You can do this by tapping your controller?s acceleration button every time the screen goes blank in between ?Get Ready? 3? 2? 1?.

• Alex Kidd (from Alex Kidd in Miracle World)

  How To Unlock: Unlocks as a present for you on Christmas Day, December 25th, 2012.
  How To Unlock Him Early: This has been confirmed on Xbox 360, PS3 & Wii U.
  Step #1: Sign out of PlayStation Network, Xbox Live, or Nintendo Network, and go to the settings menu of your console. You?ll need to change the internal clock?s date to the 25th of December in 2012 or any date further into the future.
  Step #2: Load up the game and Alex Kidd will be unlocked! You must remain offline while playing as him, or else the internet will verify the date on your machine and re-lock him.

This video guide shows how to unlock the 80?s SEGA mascot Alex Kidd early in Sonic & All-Stars Racing Transformed on Xbox 360, PS3 & Wii U. ? As far as we know it?s only playable online early in the PS3 version, the other versions you?ll have to play offline? until of course the actual date turns December 25th, 2012. Because from that date he officially unlocks forever for everyone.

• Doctor Eggman (from Sonic the Hedgehog)

  How To Unlock: Win all five mirrored Grand Prix cups.
  Tip: If you win all five standard Grand Prix cups you unlock the mirrored Grand Prix cups. And if in turn you win all five mirrored Grand Prix cups you also unlock the Expert difficulty mode.

• Metal Sonic (from Sonic the Hedgehog CD)

  How To Unlock: Metal Sonic from Sega?s Sonic the Hedgehog CD game is the first downloadable content (DLC) character. His download code (so far confirmed for Xbox 360, PlayStation 3, PlayStation Vita & Wii U) is only available to those of you who have bought the Sonic and All-Stars Racing Transformed Bonus Edition in America or the Sonic and All-Stars Racing Transformed Limited Edition in Europe.
  It is unknown if Sega intends to release Metal Sonic as free or paid DLC after the game?s launch period, previously they have not done so, but hopefully they will do it this time.

This video shows Metal Sonic racing on OutRun Bay. ? The Bonus/Limited Edition description reads it includes: ?Metal Sonic & Transforming vehicle + Metal Sonic Mod Pack, Outrun Bay Track + Challenges & Time Trials, Stickers for your in-game license.

• Future DLC Characters

  Details: On the official Sega forums, fans have started a campaign for Sega characters to be added via DLC.
  Visit this Sega forums post to find out how you can vote for your three favorites. Then after December 1st the results will be presented by the forum admin to the Sega and Sumo development team. It?s all unofficial, but if enough fans can come together, it could push Sega to add more characters to the game.

  The votes so far seem to favor:

  1. Ryo Hazuki
  2. Hatsune Miku
  3. Segata Sanshiro
  4. Vectorman
  5. Bayonetta
  6. Ristar
  7. ToeJam & Earl
  8. Billy Hatcher
  9. Axel Stone
  10. Blaze Fielding
  11. Phantom R
  12. Selvaria Bles
  13. Alis Landale
  14. Welkin Gunther
  15. Sakura Shinguuji
  16. Ash
  17. Opa-Opa
  18. Arle Nadja
  19. Kazuma Kiryu
  20. Sketch Turner
  21. The Chu Chus
  22. Space Harrier
  23. Bonanza Bros
  24. Amitie
  25. Spiral Knight
  26. Rag Rappy
  27. Jacky Bryant
  28. Ecco

  I doubt they?ll add Ristar, though. Because the development team already said he?s the Flagman instead of playable, since they have ?issues with what the character looks like from behind.? Weird, but true!

  Who could be the next DLC character?

Discoveries by: Michael, NearRiversDN, TheSonicShow, Steven & Mike

What new Sonic & All-Stars Racing Transformed characters would you like to play as?

About the author

By Ferry Groenendijk: He is the founder and editor of Video Games Blogger. He loved gaming from the moment he got a Nintendo with Super Mario Bros. on his 8th birthday. Learn more about him here and connect with him on Twitter, Facebook and at Google+.

Source: http://www.videogamesblogger.com/2012/11/28/how-to-unlock-all-sonic-all-stars-racing-transformed-characters.htm

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Smartphone Study is Good News for Apple, Bad News for Canon

Apple (NASDAQ: AAPL), Samsung, Nokia (NYSE: NOK), Research In Motion (NASDAQ: RIMM) and other smartphone manufacturers can rejoice in the news that consumers are repeatedly turning to their cellular devices to perform a wide array of tasks.

According to PCMag, 82 percent of smartphone users turn on the device just to take a picture, while 80 percent turn it on to send or receive a text message. More than half (56 percent) turn it on to surf the Web; 50 percent do so to send or receive an e-mail.

These findings are not as surprising as they are damaging to companies that produce specific-use devices. Canon (NYSE: CAJ), for example, does not make cellular devices. But it produces a number of digital cameras, including several pocket-sized devices. If Consumers are reaching for their smartphones instead of an actual camera to take photos, Canon's future could be in jeopardy.

Year-to-date Canon shares are down more than 22 percent. The company experienced a few spikes during the summer months, gaining more than 12 percent. Those gains were all but diminished as investors abandoned the stock at the end of the season. They came back in September, raising Canon shares by 14 percent. Those gains were short-lived, however. In the weeks that followed, the company lost more than 17 percent of its value.

Up until this week, Canon had been experiencing another spike, rallying 15 percent since November 14. Those gains have not been able to offset the company's overall losses, however.

At the same time, Apple's shares have been indirectly enhanced by the camera featured in the iPhone. While no single stock move can be attributed to its photographic capabilities, it is no secret that millions of consumers purchased the iPhone 5 for its camera. Despite having endured a three-month decline of more than 13 percent, Apple shares are still up more than 42 percent year-to-date.

Follow me @LouisBedigianBZ

(c) 2012 Benzinga.com. Benzinga does not provide investment advice. All rights reserved.

Tags: Apple, Canon, iPhone 5

Posted in: News, Tech, Trading Ideas, Best of Benzinga

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Source: http://www.benzinga.com/news/12/11/3127621/smartphone-study-is-good-news-for-apple-bad-news-for-canon

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Obama to host Romney at White House on Thursday (reuters)

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50 Cent And Eminem's 'My Life' Video Premieres Tuesday On MTV!

Tune in to MTV, MTV2, MTV Jams, MTV Hits and mtvU at 6 a.m. to catch the new video, featuring Maroon 5's Adam Levine on the chorus.
By Rob Markman, with reporting by Rahman Dukes

50 Cent
Photo: MTV News

Source: http://www.mtv.com/news/articles/1697949/50-cent-eminem-my-life-video-premiere-tuesday-mtv.jhtml

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Acer Aspire S3-391-6046

The Acer Aspire S3-391-6046 ($648 list) proves that you don't have to start at $899 to get a decent ultrabook. To keep the price down, it uses an older second-generation Intel Core i3 processor, but that CPU is still faster than the AMD E series APU, Intel Atom, or Celeron CPU in other inexpensive laptops. The S3-391-6046 is also an inexpensive way to introduce your family to Windows 8 and that operating system's radically new interface. It's being featured at $499 at Walmart for Black Friday 2012, but it's still a well-equipped system after the price jumps back to the $648 regular price. It's a much better laptop than the netbooks you used to expect in this price range, and as such it comes recommended.

Design and Features
The S3-391-6046 looks every bit the ultrabook, with a thin and light design in line with other Acer ultrabooks like the Acer Aspire S5-391-9880. The S3-391-6046 measures by 8.52 by 12.59 by 0.68 inches (HWD). This means that it's much thinner and lighter than the budget laptop Editors' Choice Acer Aspire V5-571-6891 ($499.99 list, 4.0 stars). Granted, the V5-571-6891 is more of a desktop replacement laptop with a larger 15-inch screen and a DVD drive, but they are priced and aimed at the same budget market.

The S3-391-6046 is more portable, with a 13.3-inch screen with a 1,366-by-768 resolution (720p). That is smaller than the 1,920 by 1,080 resolution (1080p) found on ultrabooks like the Asus Zenbook Prime UX31A-R5102F, but that's okay, since you'll pay quite a price premium for that 1080p screen, and it's not absolutely necessary at this budget price point. The 1080p resolution is more important to movie buffs and people who do a lot a multitasking with multiple windows open (like spreadsheets), and we think 1,366-by-768 is adequate for home and student use. The screen itself is bright and clear, displaying smooth text and graphics.

The S3-391-6046 weighs three pounds even, which is average for 13-inch ultrabooks, but a lot lighter than larger five-pound laptops you'd find under $500. The included AC adapter adds less than three-quarters of a pound to that in travel weight. The weight and size mean that the S3-391-6046 is a good companion for those who constantly roam the house or need to traverse a school campus. It's also well suited to be your PC companion on a vacation.

The system comes with a pair of USB 3.0 ports and no USB 2.0 ports, saving users the confusion over which port to use. The S3-391-6046 comes with a full-sized HDMI port, so you can hook the laptop up to a large HDTV if you wish. The system has a full-size QWERTY keyboard, but the arrow keys to the lower right are more compact than usual. This isn't too much of a problem for most, but it could make navigating larger spreadsheets or databases harder for those with fists of ham.

The laptop's multitouch touchpad supports all the expected pinch, zoom, scroll, and Windows 8 functions, like bringing up the Charms Bar. That is a good thing, considering that this ultrabook doesn't come with a touch screen display (nor should you expect one at this price point). The touchpad is very responsive, and it is easy to get used to moving the cursor around new Windows 8 interface. Make sure you take a look at the included Windows 8 cheat sheet if this is your first Windows 8 PC. Many of the ways you're used to doing things in previous versions of Windows have changed in Windows 8. The system comes with a 1.3-megapixel webcam for Skype and self portraits.

The S3-391-6046 comes with a bunch of pre-loaded programs (Amazon, eBay, Evernote, newsXpresso, Office 201 trial, WildTanget games, Netflix, Skype, etc.), and while they aren't obtrusive in the new Windows 8 Start screen, they do clutter up the system's desktop mode (aka the screen that looks like your old PC). Connecting the system to our 802.11 a/b/g/n Wi-Fi networks were easy, as the S3-391-6046 support both 2.4GHz and 5GHz Wi-Fi, giving you extra flexibility. The system lacks an Ethernet port, but wired Ethernet is less prevalent in the home, it's more of a must have in business offices.

The S3-391-6046 comes with a second generation Intel Core i3-2367M processor, four GB of system memory, Intel HD Graphics, a 320GB hard drive and 20GB SSD for speed. The SSD is invisible to the user, it's here to provide speed in booting, launching apps, and waking the system from sleep. It's a little slower than an SSD-only ultrabook, but SSD-only ultrabooks like the Asus Zenbook Prime cost hundreds of dollars more. The system wakes from sleep in a quick 5 seconds, which is about right for an ultrabook. The second generation Intel Core i3 processor is a little bit older than the current third generation processors found in higher end ultrabooks, but it's perfectly alright for home and school users. See more below in our performance section. The system comes with a one-year warranty, which is average for consumer laptops.

Performance
The system's Core i3 processor is much better than the Intel Celeron processor found in lesser budget systems. For example, the S3-391-6046 got a modest 1,883 point score at the PCMark 7 benchmark test, but that's still better than the Celeron-powered Toshiba Satellite C655-S5542 and Acer Aspire 5349-2635 that couldn't run PCMark 7 to competition. The faster, newer Core i3 in the Acer V5-571-6891 was able to help that system achieve a better 2,090 point score.

What's more relevant is the respective system's battery life. The S3-391-6046 comes in with a decent four hours, forty-seven minutes on our video rundown test, a little under Acer's 5.5 hour claims. This is still forty minutes longer than the V5-571-2635 on the same test. Thirteen minutes short of five hours is pretty good for a home or school bound laptop. That gives you enough power to watch a couple movies on the laptop itself, or surf while doing the same in your TV room.

If there's any drawback to using older tech in the laptop, it's that the system doesn't support DX11 3D for newer games. That said, if you have no idea what DX11 is, then you're not missing out on a lot. The system plays casual games like Cut the Rope (found in the Windows 8 Store) and Angry Birds (in your browser) just fine. Videos played smoothly on the 13.3-inch screen, and navigating around the Windows 8 interface was snappy and responsive.

The Acer Aspire S3-391-6046 is a tempting ultrabook. At the $499 Black Friday price, it's a no-brainer, as it's competent to a T with nice styling and portability. The Acer Aspire V5-571-6891 holds on to the current Editors' Choice for budget laptops, since it has a larger hard drive, more memory, a better list price, and a larger screen (with the same resolution). However, for a user who wants more portability, the S3-391-6046 makes a good second choice. If you want to dip your toe in and see if you like the Windows 8 interface, the Acer Aspire S3-391-6046 is a very good system to try.

BENCHMARK TEST RESULTS

COMPARISON TABLE
Compare the Acer Aspire S3-391-6046 with several other laptops side by side.

More Ultrabook Reviews:
??? Acer Aspire S3-391-6046
??? Lenovo IdeaPad Yoga 13
??? Acer Aspire S7-391-9886
??? HP Envy TouchSmart Ultrabook 4t-1100
??? Dell XPS 12
?? more

Source: http://feedproxy.google.com/~r/ziffdavis/pcmag/~3/J0k4a-wQ6p0/0,2817,2412387,00.asp

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Hungarian Jews ask Russia to return looted scrolls

PRAGUE (Reuters) - Hungarian Jews have asked Russia to return scrolls and valuable religious items looted by Nazis and the Red Army in World War Two and their claim has been backed by the Budapest government.

Some 300 to 400 Torah scrolls and thousands of vestments, crowns and other articles are being held in museums and storage in Russia, said Slomo Koves, executive rabbi of the Unified Hungarian Jewish Congregation.

Seized by Nazi Germany during the war and then by the Red Army in 1945, many of the items had been stored for safety in the Hungarian National Bank during the Holocaust.

Up to 600,000 Jews died at the hands of the Nazis and their allies, according to Budapest's Holocaust Memorial Centre, and Hungary's Jewish community, now numbering around 100,000, still faces anti-Semitism.

On Tuesday, a far-right politician urged the government to draw up list of Jews who posed a "national security risk".

In June, Nobel Peace Prize laureate Elie Wiesel repudiated a Hungarian state award he received after an official from the Hungarian government participated in a ceremony honoring a writer who was a member of Hungary's WW2 far-right parliament.

Koves said he had sent a letter to Russian authorities about the religious artifacts, which are part of a larger body of art and valuables that Hungarian authorities say the Soviets took from the country when the war ended.

"Our request is that if we're not speaking about looted art, at least we can speak about all the pieces that are important to the Jewish community," Koves said.

He had asked for Moscow to allow a delegation to document the items and then work out a process for their return.

Hungarian Foreign Minister Janosz Martonyi has also written to Moscow to say the issue had been "unfortunately dragging on for a long time now" and saying it was "of great national importance for the whole country".

Russia's Foreign Ministry declined immediate comment.

About one tenth of the 6 million Jews who were killed during the Holocaust came from Hungary. Most were deported to death camps after Nazi Germany occupied the country in 1944, often after soldiers seized their possessions.

Historians have documented stories of people who risked their lives or died trying to protect Torah scrolls. Some who ran into burning synagogues or hid them on their person when they were deported to death camps.

SCROLLS IN MUSEUMS

Torah scrolls are at the heart of Jewish worship, and a community needs to obtain them before it builds a synagogue.

Painstakingly copied by hand on parchment, they contain the five books of Moses and are kept in an arch of a synagogue, usually facing east towards Jerusalem. Koves said the ones in question were hundreds of years old and "priceless".

According to a catalogue compiled by the Conference on Jewish Material Claims against Germany, 344 Torah scrolls were turned over by Russia's Special Archive, now part of the Russian State Military Archive, to the State Historical Museum.

The Hungarian group said Russia also holds Torahs in the Nizhny Novgorod Museum. Some scholars say these originated in Hungary while others say they came from Russian synagogues.

"When we speak of Torah scrolls, there is no question that they belong to the Jewish community, and we are all heirs of the survivors," Koves said.

"Today, when we don't have to endanger our lives but just have to bring it to people's attention, this is our first duty as Hungarian Jews." Some 8,000 Jewish Holocaust survivors live in Hungary.

(Additional reporting by Krisztina Than and Martin Dunai in Budapest; editing by Robert Woodward)

Source: http://news.yahoo.com/hungarian-jews-ask-russia-return-looted-scrolls-135442822.html

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Russian pro-business party plans legalization of prostitution- leader ...

Andrey Dunayev (RIA Novosti / Iliya Pitalev)

Right Cause leader Andrey Dunayev says the party has started preparing opinion polls in order to understand the people?s attitude to the problem of prostitution.

?We are not promoting easier access to prostitutes, but want to attract attention to the problem, because these women work in absolutely intolerable conditions, they are used as slaves and later they are thrown into the street, sick and wanted by nobody,? Dunayev said in an interview with Izvestia daily.

He added that the party has already contacted the leading polling services in order to start questioning citizens. The research itself will start in January 2013, the politician promised.

The Right Cause insists prostitutes must be granted the same rights as the rest of the country?s citizens. Authorities must allow the opening of official brothels, allow unions and open work for prostitutes. These women must undergo regular medical checks and be granted official pensions upon retirement, Dunayev explained.

However, the head of the VTSIOM polling service, Valeriy Fyodorov, says Russian society still has very negative attitude to prostitution. The researcher said the problem needed serious research and added that his company was ready to participate in it.

Dunayev?s interview was Just Cause?s first appearance in the media in months. Previous reports said that in a bid for popularity the party attracted flamboyant priest-turned-actor Ivan Okhlobystin, but he left quickly on the order of church officials.

Before that, the reports about the party mostly covered the scandals it got involved in. The most notable is the 2011 elections campaign when Right Cause approved billionaire Mikhail Prokhorov as its leader but ousted him after the very first electoral convention.

One of the party leaders, the grandmaster of the all-Russian freemason lodge Andrey Bogdanov, led the veterans? rebellion against the billionaire protesting against Prokhorov?s supposedly authoritarian management style. Prokhorov left, but demanded that the party returned the money already spent on its promotion. The Right Cause complied but failed to win a single seat in the parliamentary election.

Since then, Bogdanov himself left the Right Cause for the re-registered Democratic Party of Russia. He sharply criticized his former colleagues over the idea of prostitution legalization saying that the conservative politicians and religious people cannot welcome such initiatives. He promised to oppose both the calling of the referendum and the legalization approval and expressed doubt that the Right Cause is capable of collecting one million signatures required for the referendum initiating.

Source: http://rt.com/politics/right-plans-prostitution-leader-603/

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Australia apologizes to military victims of abuse - tonight Newspaper ...

Australia apologizes to military victims of abuse

26 November 2012 No Comment

Rod McGuirk, The Associated Press | Last Updated:Mon, 26 Nov 2012 07:10:56 GMT

CANBERRA, Australia - Australia's defence minister on Monday apologized to military personnel past and present who were sexually abused or otherwise mistreated during their service. He also started an inquiry into hundreds of allegations of abuse over six decades.

Defence Minister Stephen Smith made the apology in Parliament on behalf of the government in the latest step in a two-year effort to reform the culture of the Australian military and it make more accepting of women.

"Young men and women have suffered treatment which no member of our defence force or our community generally should experience," Smith said.

"Young men and women have endured sexual, physical or mental abuse from their colleagues which are not acceptable and do not reflect the values of a modern, diverse, tolerant, Australian society," he added.

He noted claims that officers had abused their positions of trust through their own behaviour or by turning a blind eye to the actions of others.

Smith also announced that retired judge Len Roberts-Smith had been appointed to examine allegations of abuse by more than 1,000 alleged victims across every decade since 1950s.

The earliest case relates to the alleged abuse of a 13-year-old navy trainee in 1951, while the most recent relates to events in 2011.

A preliminary review of these allegations by a law firm found that 750 were "plausible," Smith said.

The three-month inquiry could result in compensation of up to 50,000 Australian dollars ($52,000) for each victim and the alleged perpetrators being referred to criminal authorities for prosecution.

Smith said some of the perpetrators could still be serving in the military.

The government started inquiries last year in response to a young woman's allegation that a fellow cadet had secretly filmed a sexual encounter between the pair and broadcast it to their colleagues at the Australian military officer training academy. The incident and the attention the government focused on it provoked a wave of complaints of sexual misconduct over the decades.

Source: http://www.tonightnewspaper.com/2012/11/26/australia-apologizes-to-military-victims-of-abuse/

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China's role in Southeast Asia questioned

FILE - In this Nov. 20, 2012 file photo, China's Premier Wen Jiabao, right, chats with U.S. President Barack Obama, left, as Cambodia's Prime Minister Hun Sen stands between them during a family photo at the 7th East Asia Summit Plenary Session in Phnom Penh, Cambodia. China's finding the once friendly ground of Southeast Asia bumpy going, with anger against Chinese claims to disputed islands, once reliable ally Myanmar flirting with democracy and renewed American attention to the region. (AP Photo/Vincent Thian, File)

FILE - In this Nov. 20, 2012 file photo, China's Premier Wen Jiabao, right, chats with U.S. President Barack Obama, left, as Cambodia's Prime Minister Hun Sen stands between them during a family photo at the 7th East Asia Summit Plenary Session in Phnom Penh, Cambodia. China's finding the once friendly ground of Southeast Asia bumpy going, with anger against Chinese claims to disputed islands, once reliable ally Myanmar flirting with democracy and renewed American attention to the region. (AP Photo/Vincent Thian, File)

FILE - In this Nov. 19, 2012 file photo, China's Premier Wen Jiabao, second left, joins hands with South Korea's President Lee Myung-bak, left, Cambodia's Prime Minister Hun Sen, third lfrom eft, and Japan's Prime Minister Yoshihiko Noda during the ASEAN Plus Three (APT) Commemorative Summit in Phnom Penh, Cambodia. China's finding the once friendly ground of Southeast Asia bumpy going, with anger against Chinese claims to disputed islands, once reliable ally Myanmar flirting with democracy and renewed American attention to the region. (AP Photo/Vincent Thian, File)

FILE - In this Nov. 5, 2012 file photo, China's Premier Wen Jiabao arrives for the opening ceremony of the ASEM Summit in Vientiane, Laos. China's finding the once friendly ground of Southeast Asia bumpy going, with anger against Chinese claims to disputed islands, once reliable ally Myanmar flirting with democracy and renewed American attention to the region. (AP Photo/Vincent Thian, File)

BEIJING (AP) ? China is finding the once friendly ground of Southeast Asia bumpy going, with anger against Chinese claims to disputed islands, once reliable ally Myanmar flirting with democracy and renewed American attention to the region.

The changing terrain for Beijing was on view this past week at a conclave of East Asian nations in Cambodia. Wen Jiabao, China's lame duck premier who usually exudes a mild, grandfatherly air, got into a sharp exchange over the contested South China Sea islands. The leaders of the Philippines, Singapore and Vietnam reacted furiously when host Cambodia suggested that all sides agreed not to bring outside parties into the dispute ? a reference to the U.S.

Meanwhile, Barack Obama, buoyed by the first visit ever by a U.S. president to Myanmar, projected an image of a confident, friendly America, calling for a reduction in tensions and seemingly taking no sides.

Beijing is struggling to find its feet as its own power grows, but the U.S. refuses to cede influence in the region, emboldening other countries not to fall in with the Chinese line.

"The robust U.S. presence and relatively disciplined and quiet diplomacy looked strong relative to China's heavy-handed pressure," Ernest Bower, chair for Southeast Asian studies at the Council for Strategic and International Studies in Washington, D.C., wrote in a Thursday commentary.

It's a reversal over the treatment Beijing enjoyed much of the past decade as it wooed Southeast Asia with soaring trade and investment and the lure of the huge Chinese market. Looking to further those links, Wen held discussions on expanding a free trade agreement to increase China's imports from Southeast Asia.

China's economic "pull remains, but the smile has faded," said Aaron Friedberg, professor of politics and international affairs at Princeton University.

Getting Southeast Asian diplomacy right matters to Beijing. It's an area where China historically exercised great sway. The 10 countries of the Association of Southeast Asian nations, or ASEAN, are home to a market of 600 million people and straddle vital shipping lanes and seas rich in fish, oil, gas and other minerals.

Beijing's influence began foundering in 2010 when its more assertive claims to islands in the South China Sea touched off anxieties among the Philippines and Vietnam, who along with Brunei, Malaysia and Taiwan also claim the islands in whole or in part.

The fracas provided an opening for the U.S., which as it wound down involvement in Iraq was re-examining the challenge posed by China. The U.S. "pivot" brought renewed diplomatic attention to the region and promises of more military resources.

Still, the friction has only increased. Beijing has become more aggressive in patrolling around the disputed islands, leading to a faceoff last summer with the Philippines over Scarborough Shoal. It is sparring farther afield over other islands with Japan, heightening worries about an expansionist China. It also started issuing new passports featuring a map that shows the entire South China Sea as Chinese territory.

The tensions bubbled to the fore at an annual summit of Southeast Asian leaders in Cambodia's capital of Phnom Penh attended by Obama.

Philippine President Benigno Aquino raised the Scarborough Shoal, prompting Chinese Premier Wen Jiabao to state that the islets have been "Chinese territory since ancient times and no sovereignty dispute exists." China's actions to assert its sovereignty were wholly "appropriate and necessary," Wen told the closed door meeting, according to Vice Foreign Minister Fu Ying.

Wen's stern statement was "destructive and dangerous," wrote CSIS's Bower. "This is very uncertain ground, and uncertainty means the emergency of an inherent instability in the region that undermines a solid foundation for regional growth."

Chinese government-backed experts conceded a failure in execution. "Somehow, the issue was not handled very well in the meeting," said Zhao Gancheng, director of the Center for Southeast Asia at the Shanghai Institute for Foreign Studies.

Economic realities could still work in China's favor, experts say. Chinese imports from the region grew 29 percent last year to $146 billion, and with its economy expected to overtake America's as the world's largest in coming years, China will only grow in importance as a source of overseas investment.

The very fact that China has refused to back off ? despite provoking a backlash that could hurt its long-term interests ? speaks to Beijing's belief that its economic pull will ultimately convince its ASEAN neighbors that their future lies with China, not with the U.S., said Princeton's Friedberg.

"The big question, I think, is whether the ASEAN states believe that the United States actually has the resolve and the resources to follow through on the commitments that have been made in recent years. If they begin to doubt this they will have to do more to appease Beijing," Friedberg said.

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/cae69a7523db45408eeb2b3a98c0c9c5/Article_2012-11-25-China-Southeast%20Asia/id-920a6a2fc3aa4a82b71158a3e5ad0bc6

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Local Group Provides Support, Education and Advocacy for Prostate ...

By Myron Thompson

Celebrate, remember, fight back, hope and cure are key words for all who have been touched with the dreaded word ?cancer?. For men, the words ?You have prostate cancer? strikes fear or great concern to those given the diagnosis.? Today, such devastating news means it is time to seek a cure, fight back as a warrior, and reach out for support in the new journey awaiting.

The Tacoma Prostate Cancer Support Group provides a community of caring individuals who willingly give support and education to the recently diagnosed person and those with an on-going battle with the disease.? Information, books, and pamphlets on prostate cancer are available for free at the twice monthly meetings held in Tacoma.

?

Jack Hudspeth, support group facilitator, says that one extremely important aspect of the disease is to get tested early with a PSA test.? Hudspeth said of the PSA test, ?It?s the gold standard of prostate tests today?.? He further adds, ?While?new tests are coming, this is the best we have today?.

Hudspeth is very adamant that the simple PSA blood test saved his life as it has with countless other men.? He was diagnosed in 1997 with a rising PSA level.? Further tests revealed he had aggressive prostate cancer, and without treatment he was headed for an early demise.? He is now in his 70?s and walking laps at the 2012 Tacoma Relay for Life and participating in the annual Sound to Narrows walk.? In October this charter member of the Tacoma Prostate Cancer Support Group hiked Mt. Si to raise money for prostate cancer awareness.

Tom Kirk, CEO of the UsTOO International Prostate Cancer Education and Support Network, mentioned at a recent Town Hall Meeting in Tacoma that men in their 40?s are being diagnosed prostate cancer.? Many of these men whose cancer is caught too late will lose their lives.? With early detection, most of these men would survive.? Mr. Kirk addressed the recent US Task Force Report on Prostate Cancer Screening whose recommendation is to eliminate the PSA test for most men saying it is not needed.? This same group recommended doing away with early mammograms for women.? Now the PSA test for men is under fire from the same group.

Willie Stewart, also a facilitator, is a former teacher and administrator with the Tacoma Public Schools.? He was instrumental in starting the Tacoma Prostate Cancer Support Group then known as Man To Man in 1990.? Stewart remembers vividly the day he was exercising at the YMCA when he was urged to get a new test called the PSA.? He decided to get tested which led to a diagnosis of prostate cancer.? Early detection and surgery saved his life.? As a man of African-American heritage, Stewart understands that men of his race are more prone to get prostate cancer than other races.? Stewart prides himself in attending community health fairs to get his message heard.

Bob Freeborn is a current facilitator and chairperson of the Tacoma Prostate Cancer Support Group Steering Committee.? He along with Jack Hudspeth and Willie Stewart invite noted health professionals from the greater Puget Sound Region to speak on the many aspects of prostate cancer and the resources available in the area.

The public is invited to the support group meetings held on the second Thursday and fourth Tuesday of each month.? The Thursday meeting is a round table discussion addressing the newly diagnosed and/or those whose PSA is rising.? The Tuesday meeting brings in experts to present their views and knowledge of prostate cancer and related health topics.? Meetings are held at the University Place Presbyterian Church located at 8101 27^th?St. West, and start promptly at 6:00 PM. The next meeting will be held on Tuesday, November 27, 2012.? The? group does not meet in December. Men and women are encouraged to attend.? There is no charge.

For further information go to?www.tpcsg.info., or call Bob Freeborn at (253) 566-1651.

The unanimous cry goes out from TPCSG members, ?Get the PSA test and become informed! It can help save your life.?

Category: Announcements

Source: http://www.thesubtimes.com/local-group-provides-support-education-and-advocacy-for-prostate-cancer/?utm_source=rss&utm_medium=rss&utm_campaign=local-group-provides-support-education-and-advocacy-for-prostate-cancer

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Wild Birds Unlimited: The family tree of all birds

Image by Arne Mooers (SFU),

Gavin Thomas (Sheffield) and Cody Shrank (Yale

A Yale-led scientific team has produced the most comprehensive family tree for birds connecting all living bird species and revealing surprising new details about their evolutionary history and its geographic context.

The researchers relied heavily on fossil and DNA data, combining them with geographical information to produce the exhaustive family tree, which includes 9,993 species known to be alive now. Analysis of the family tree shows when and where birds diversified ? and that birds? diversification rate has increased over the last 50 million years, challenging the conventional wisdom of biodiversity experts.

??The current zeitgeist in biodiversity science is that the world can fill up quickly,? says biologist and co-author Arne Mooers of Simon Fraser University in Canada. ?A new distinctive group, like bumblebees or tunafish, first evolves, and, if conditions are right, it quickly radiates to produce a large number of species.?These species fill up all the available niches, and then there is nowhere to go. Extinction catches up, and things begin to slow down or stall.?For birds the pattern is the opposite: Speciation is actually speeding up, not slowing down.??

The researchers attribute the growing rate of avian diversity to an abundance of group-specific adaptations. They hypothesize that the evolution of physical or behavioral innovations in certain groups, combined with the opening of new habitats, has enabled repeated bursts of diversification. Another likely factor has been birds? exceptional mobility, researchers said, which time and again has allowed them to colonize new regions and exploit novel ecological opportunities.

Source:?
The global diversity of birds in space and time
W. Jetz, G. H. Thomas, J. B. Joy, K. Hartmann, A. O. Mooers doi:10.1038/nature11631,
URL: http://www.nature.com/nature/journal/v491/n7424/full/nature11631.html

Source: http://lansingwbu.blogspot.com/2012/11/the-family-tree-of-all-birds.html

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ScienceDaily: Gene News

http://www.sciencedaily.com/news/health_medicine/genes/ Genes and Genetics News. Read today's medical research in genetics including what can damage genes, what can protect them, and more.en-usSat, 24 Nov 2012 17:44:48 ESTSat, 24 Nov 2012 17:44:48 EST60http://www.sciencedaily.com/images/logosmall.gifhttp://www.sciencedaily.com/news/health_medicine/genes/ For more science articles, visit ScienceDaily.http://www.sciencedaily.com/releases/2012/11/121124090511.htm Researchers have uncovered a protein ?partner? commonly used by breast cancer cells to unlock genes needed for spreading the disease around the body. A report on the discovery details how some tumors get the tools they need to metastasize.Sat, 24 Nov 2012 09:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121124090511.htmhttp://www.sciencedaily.com/releases/2012/11/121123092132.htm The genome encodes the complete information needed by an organism, including that required for protein production. Viruses, which are up to a thousand times smaller than human cells, have considerably smaller genomes. Using a type of herpesvirus as a model system scientists have shown that the genome of this virus contains much more information than previously assumed. The researchers identified several hundred novel proteins, many of which were surprisingly small.Fri, 23 Nov 2012 09:21:21 ESThttp://www.sciencedaily.com/releases/2012/11/121123092132.htmhttp://www.sciencedaily.com/releases/2012/11/121122152928.htm Scientists have made a major advance in understanding how flu viruses replicate within infected cells. The researchers used cutting-edge molecular biology and electron-microscopy techniques to ?see? one of influenza?s essential protein complexes in unprecedented detail. The images generated in the study show flu virus proteins in the act of self-replication, highlighting the virus?s vulnerabilities that are sure to be of interest to drug developers.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152928.htmhttp://www.sciencedaily.com/releases/2012/11/121122152910.htm Fifty years after scientists first posed a question about protein folding, the search for answers has led to the creation of a full-fledged field of research that led to major advances in supercomputers, new materials and drug discovery, and shaped our understanding of the basic processes of life, including so-called "protein-folding diseases" such as Alzheimer's, Parkinson's and type II diabetes.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152910.htmhttp://www.sciencedaily.com/releases/2012/11/121121145638.htm Researchers recently uncovered a nerve cell's internal clock, used during embryonic development. This breakthrough could lead to the development of new tools to repair and regenerate nerve cells following injuries to the central nervous system.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145638.htmhttp://www.sciencedaily.com/releases/2012/11/121121145621.htm Using a new technique called cryo-electron tomography, scientists have created a three-dimensional map that gives a better understanding of how the architecture of the rod sensory cilium (part of one type of photoreceptor in the eye) is changed by genetic mutation and how that affects its ability to transport proteins as part of the light-sensing process.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145621.htmhttp://www.sciencedaily.com/releases/2012/11/121121130933.htm Researchers have resolved the structure of that allows a telomere-related protein, Cdc13, to form dimers in yeast. Mutations in this region of Cdc13 put the kibosh on the ability of telomerase and other proteins to maintain telomeres.Wed, 21 Nov 2012 13:09:09 ESThttp://www.sciencedaily.com/releases/2012/11/121121130933.htmhttp://www.sciencedaily.com/releases/2012/11/121121130811.htm Cancer therapies often have short-lived benefits due to the emergence of genetic mutations that cause drug resistance. A key gene that determines resistance to a range of cancer drugs has been reported in a new study. The study reveals a biomarker that can predict responses to cancer drugs and offers a strategy to treat drug-resistant tumors based on their genetic signature.Wed, 21 Nov 2012 13:08:08 ESThttp://www.sciencedaily.com/releases/2012/11/121121130811.htmhttp://www.sciencedaily.com/releases/2012/11/121121130703.htm Two recent studies delve into the role of chromatin modifying enzymes and transcription factors in tumour cells. In one, it was found that the PARP1 enzyme activated by kinase CDK2 is necessary to induce the genes responsible for the proliferation of breast cancer cells in response to progesterone. In another, extensive work has been undertaken to identify those genes activated by the administration of progesterone in breast cancer, the sequences that can be recognized and how these genes are induced.Wed, 21 Nov 2012 13:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121121130703.htmhttp://www.sciencedaily.com/releases/2012/11/121121130643.htm Previously discarded, human-specific ?junk? DNA represents untapped resource in the study of diseases like Alzheimer?s and autism.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130643.htmhttp://www.sciencedaily.com/releases/2012/11/121121130633.htm In a new study, researchers describe markers and a model that quantify how aging occurs at the level of genes and molecules, providing not just a more precise way to determine how old someone is, but also perhaps anticipate or treat ailments and diseases that come with the passage of time.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130633.htmhttp://www.sciencedaily.com/releases/2012/11/121121104552.htm New research has found there are several different ways that kidney tumors can achieve the same result -- namely, grow.Wed, 21 Nov 2012 10:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121121104552.htmhttp://www.sciencedaily.com/releases/2012/11/121121104416.htm Clumped proteins can be dissolved with the aid of cellular repair systems -- a process of critical importance for cell survival especially under conditions of stress. Researchers have now decrypted the fundamental mechanism for dissolving protein aggregates that involves specific molecular chaperones.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104416.htmhttp://www.sciencedaily.com/releases/2012/11/121121104401.htm New understanding of molecular changes that convert harmless cells surrounding ovarian cancer cells into cells that promote tumor growth and metastasis provides potential new therapeutic targets for this deadly disease, according to new research.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104401.htmhttp://www.sciencedaily.com/releases/2012/11/121120194932.htm A new rapid test for tuberculosis (TB) could substantially and cost-effectively reduce TB deaths and improve treatment in southern Africa -- a region where both HIV and tuberculosis are common.Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194932.htmhttp://www.sciencedaily.com/releases/2012/11/121120194926.htm A new study has identified hundreds of small regions of the genome that appear to be uniquely regulated in human neurons. These regulatory differences distinguish us from other primates, including monkeys and apes, and as neurons are at the core of our unique cognitive abilities, these features may ultimately hold the key to our intellectual prowess (and also to our potential vulnerability to a wide range of 'human-specific' diseases from autism to Alzheimer's).Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194926.htmhttp://www.sciencedaily.com/releases/2012/11/121120132906.htm Rather than target RNA viruses directly, aiming at the host cells they invade could hold promise, but any such strategy would have to be harmless to the host. Now, a surprising discovery made in ribosomes may point the way to fighting fatal viral infections such as rabies.Tue, 20 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121120132906.htmhttp://www.sciencedaily.com/releases/2012/11/121120121835.htm Microbiologists studying bacterial colonization in mice have discovered how the very rapid and efficient spread of antibiotic resistance works in the respiratory pathogen, Streptococcus pneumoniae (also known as the pneumococcus). The team found that resistance stems from the transfer of DNA between bacterial strains in biofilms in the nasopharynx, the area just behind the nose.Tue, 20 Nov 2012 12:18:18 ESThttp://www.sciencedaily.com/releases/2012/11/121120121835.htmhttp://www.sciencedaily.com/releases/2012/11/121120100155.htm Scientists have discovered another molecule that plays an important role in regulating myelin formation in the central nervous system. Myelin promotes the conduction of nerve cell impulses by forming a sheath around their projections, the so-called axons, at specific locations -- acting like the plastic insulation around a power cord.Tue, 20 Nov 2012 10:01:01 ESThttp://www.sciencedaily.com/releases/2012/11/121120100155.htmhttp://www.sciencedaily.com/releases/2012/11/121120084725.htm Researchers have discovered new genetic evidence about why some people are happier than others. The scientists have uncovered evidence that the gene FTO -- the major genetic contributor to obesity -- is associated with an eight per cent reduction in the risk of depression. In other words, it's not just an obesity gene but a "happy gene" as well.Tue, 20 Nov 2012 08:47:47 ESThttp://www.sciencedaily.com/releases/2012/11/121120084725.htmhttp://www.sciencedaily.com/releases/2012/11/121119213144.htm Researchers have found that biological age and life expectancy can be predicted by measuring an individual's DNA. They studied the length of chromosome caps -- known as telomeres -- in a 320-strong wild population of Seychelles Warblers on a small isolated island.Mon, 19 Nov 2012 21:31:31 ESThttp://www.sciencedaily.com/releases/2012/11/121119213144.htmhttp://www.sciencedaily.com/releases/2012/11/121119171403.htm Certain mutated cells keep trying to replicate their DNA -- with disastrous results -- even after medications rob them of the raw materials to do so, according to new research.Mon, 19 Nov 2012 17:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121119171403.htmhttp://www.sciencedaily.com/releases/2012/11/121119163301.htm DNA sequences obtained from a handful of patients with multiple sclerosis have revealed the existence of an ?immune exchange? that allows the disease-causing cells to move in and out of the brain.Mon, 19 Nov 2012 16:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121119163301.htmhttp://www.sciencedaily.com/releases/2012/11/121119132056.htm Changes in how DNA interacts with histones ?- the proteins that package DNA ?- regulate many fundamental cell activities from stem cells maturing into a specific body cell type or blood cells becoming leukemic. These interactions are governed by a biochemical tug of war between repressors and activators, which chemically modify histones signaling them to clamp down tighter on DNA or move aside and allow a gene to be expressed.Mon, 19 Nov 2012 13:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121119132056.htmhttp://www.sciencedaily.com/releases/2012/11/121119114249.htm A new tool for neuroscientists delivers a thousand pinpricks of light to individual neurons in the brain. The new 3-D "light switch", created by biologists and engineers, could one day be used as a neural prosthesis that could treat conditions such as Parkinson's and epilepsy by using gene therapy to turn individual brain cells on and off with light.Mon, 19 Nov 2012 11:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121119114249.htmhttp://www.sciencedaily.com/releases/2012/11/121119104944.htm A large-scale international study has just discovered a gene for susceptibility to a rare disease providing evidence of the heterogeneous aetiology of Alzheimer's disease.Mon, 19 Nov 2012 10:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121119104944.htmhttp://www.sciencedaily.com/releases/2012/11/121119093848.htm The cerebral cortex is the most complex structure in our brain and the seat of consciousness, emotion, motor control and language. In order to fulfill these functions, it is composed of a diverse array of nerve cells, called cortical neurons, which are affected by many neurological and neuropsychiatric diseases. Researchers have opened new perspectives on brain development and stem cell neurobiology by discovering a gene called BCL6 as a key factor in the generation of cortical neurons during embryonic brain development.Mon, 19 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121119093848.htmhttp://www.sciencedaily.com/releases/2012/11/121118141530.htm Scientists have demonstrated that induced pluripotent stem cells -- the embryonic-stem-cell look-alikes whose discovery a few years ago won this year's Nobel Prize in medicine -- are not as genetically unstable as was thought.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141530.htmhttp://www.sciencedaily.com/releases/2012/11/121118141524.htm The prevailing wisdom has been that every cell in the body contains identical DNA. However, a new study of stem cells derived from the skin has found that genetic variations are widespread in the body's tissues, a finding with profound implications for genetic screening.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141524.htmhttp://www.sciencedaily.com/releases/2012/11/121118141432.htm Geneticists, pediatricians, surgeons and epidemiologists have identified two areas of the human genome associated with the most common form of non-syndromic craniosynostosis premature closure of the bony plates of the skull.Sun, 18 Nov 2012 14:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121118141432.htmhttp://www.sciencedaily.com/releases/2012/11/121117184658.htm A new discovery concerning a fundamental understanding about how DNA works will produce a "180-degree change in focus" for researchers who study how gene packaging regulates gene activity, including genes that cause cancer and other diseases.Sat, 17 Nov 2012 18:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121117184658.htmhttp://www.sciencedaily.com/releases/2012/11/121116161059.htm Adverse side effects of certain hepatitis C medications can now be replicated in the lab, thanks to a research team. The new method aids understanding of recent failures of hepatitis C antiviral drugs in some patients, and could help to identify medications that eliminate adverse effects. The findings may aid the development of safer and more effective treatments for hepatitis C and other pathogens such as SARS and West Nile virus.Fri, 16 Nov 2012 16:10:10 ESThttp://www.sciencedaily.com/releases/2012/11/121116161059.htmhttp://www.sciencedaily.com/releases/2012/11/121116124644.htm Researchers have found that a protein, known for causing cancer cells to spread around the body, is also one of the molecules that trigger repair processes in the brain.Fri, 16 Nov 2012 12:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121116124644.htmhttp://www.sciencedaily.com/releases/2012/11/121116124551.htm New research shows that a gene is responsible for a person's tendency to be an early riser or night owl -- and helps determine the time of day a person is most likely to die.Fri, 16 Nov 2012 12:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121116124551.htmhttp://www.sciencedaily.com/releases/2012/11/121116091226.htm Insights from a genetic condition that causes brain cancer are helping scientists better understand the most common type of brain tumor in children.Fri, 16 Nov 2012 09:12:12 ESThttp://www.sciencedaily.com/releases/2012/11/121116091226.htmhttp://www.sciencedaily.com/releases/2012/11/121115210541.htm Researchers have pieced together new genetic clues to the arthritis puzzle in a study that brings potential treatments closer to reality and could also provide insights into why more women than men succumb to the disabling condition.Thu, 15 Nov 2012 21:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121115210541.htmhttp://www.sciencedaily.com/releases/2012/11/121115172255.htm Passing one's genes on to the next generation is a mark of evolutionary success. So it makes sense that the body would work to ensure that the genes the next generation inherits are exact replicas of the originals. Biologists have now identified one way the body does exactly that.Thu, 15 Nov 2012 17:22:22 ESThttp://www.sciencedaily.com/releases/2012/11/121115172255.htmhttp://www.sciencedaily.com/releases/2012/11/121115152655.htm Researchers report that an efficient, high-volume technique for testing potential drug treatments for Alzheimer's disease uncovered an organic compound that restored motor function and longevity to fruit flies with the disease.Thu, 15 Nov 2012 15:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115152655.htmhttp://www.sciencedaily.com/releases/2012/11/121115133414.htm For the past several years, research has focused on the intricate actions of an ancient family of catalytic enzymes that play a key role in translation, the process of producing proteins. In a new study, scientists have shown that this enzyme can actually also work in another fundamental process in humans.Thu, 15 Nov 2012 13:34:34 ESThttp://www.sciencedaily.com/releases/2012/11/121115133414.htmhttp://www.sciencedaily.com/releases/2012/11/121115133312.htm Researchers have discovered that tanshinones, which come from the plant Danshen and are highly valued in Chinese traditional medicine, protect against the life-threatening condition sepsis.Thu, 15 Nov 2012 13:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121115133312.htmhttp://www.sciencedaily.com/releases/2012/11/121115132903.htm Medical researchers have for the first time described the structure of the active site core of topoisomerase II alpha, an important target for anti-cancer drugs. The type II topoisomerases are important enzymes that are involved in maintaining the structure of DNA and chromosome segregation during both replication and transcription of DNA. One of these enzymes, topoisomerase II alpha, is involved in the replication of DNA and cell proliferation, and is highly expressed in rapidly dividing cancer cells.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132903.htmhttp://www.sciencedaily.com/releases/2012/11/121115132901.htm Enzyme hunters at UiO have discovered the function of an enzyme that is important in the spreading of cancer. Cancer researchers now hope to inhibit the enzyme.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132901.htmhttp://www.sciencedaily.com/releases/2012/11/121115132613.htm New research has revealed a genetic link in pregnant moms - and their male partners - to pre-eclampsia, a life-threatening complication during pregnancy.Thu, 15 Nov 2012 13:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115132613.htmhttp://www.sciencedaily.com/releases/2012/11/121115132344.htm Thanks to some careful detective work, scientist better understand just how iPS cells form ? and why the Yamanaka process is inefficient, an important step to work out for regenerative medicine. The findings uncover cellular impediments to iPS cell development that, if overcome, could dramatically improve the efficiency and speed of iPS cell generation.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132344.htmhttp://www.sciencedaily.com/releases/2012/11/121115132342.htm About 10 percent of kids born with kidney defects have large alterations in their genomes known to be linked with neurodevelopmental delay and mental illness, a new study has shown.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132342.htmhttp://www.sciencedaily.com/releases/2012/11/121114172833.htm Relatively small levels of exposure to alcohol while in the womb can influence a child's IQ, according to a new study using data from over 4,000 mothers and their children.Wed, 14 Nov 2012 17:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121114172833.htmhttp://www.sciencedaily.com/releases/2012/11/121114171710.htm A gene so powerful it nearly triples the risk of Alzheimer's disease has been discovered by an international team of researchers. It is the most potent genetic risk factor for Alzheimer's identified in the past 20 years.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171710.htmhttp://www.sciencedaily.com/releases/2012/11/121114171708.htm For the first time, researchers have landed on a potential diagnostic method to identify at least a subset of patients with chronic fatigue syndrome - testing for antibodies linked to latent Epstein-Barr virus reactivation.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171708.htmhttp://www.sciencedaily.com/releases/2012/11/121114153227.htm In laboratory experiments, scientists have eliminated metastasis, the spread of cancer from the original tumor to other parts of the body, in melanoma by inhibiting a protein known as melanoma differentiation associated gene-9 (mda-9)/syntenin.Wed, 14 Nov 2012 15:32:32 ESThttp://www.sciencedaily.com/releases/2012/11/121114153227.htmhttp://www.sciencedaily.com/releases/2012/11/121114134512.htm Researchers provide a whole-genome sequence and analysis of number of pig breeds, including a miniature pig that serves a model for human medical studies and therapeutic drug testing.Wed, 14 Nov 2012 13:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121114134512.htmhttp://www.sciencedaily.com/releases/2012/11/121114134054.htm Caterpillar fungi are rare parasites found on hibernating caterpillars in the mountains of Tibet. For centuries they have been highly prized as a traditional Chinese medicine - just a small amount can fetch hundreds of dollars.Wed, 14 Nov 2012 13:40:40 ESThttp://www.sciencedaily.com/releases/2012/11/121114134054.htmhttp://www.sciencedaily.com/releases/2012/11/121114113803.htm There are plenty of effective anticancer agents around. The problem is that, very often, they cannot gain access to all the cells in solid tumors. A new gene delivery vehicle may provide a way of making tracks to the heart of the target.Wed, 14 Nov 2012 11:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121114113803.htmhttp://www.sciencedaily.com/releases/2012/11/121114113235.htm New research has uncovered the cause of infertility for 80 per cent of couples previously diagnosed with 'unexplained infertility': high sperm DNA damage.Wed, 14 Nov 2012 11:32:32 ESThttp://www.sciencedaily.com/releases/2012/11/121114113235.htmhttp://www.sciencedaily.com/releases/2012/11/121114083928.htm The ability of cannabis to produce psychosis has long been an important public health concern. This concern is growing in importance as there is emerging data that cannabis exposure during adolescence may increase the risk of developing schizophrenia, a serious psychotic disorder. Further, with the advent of medical marijuana, a new group of people with uncertain psychosis risk may be exposed to cannabis.Wed, 14 Nov 2012 08:39:39 ESThttp://www.sciencedaily.com/releases/2012/11/121114083928.htmhttp://www.sciencedaily.com/releases/2012/11/121113214635.htm For the first time, researchers have used DNA sequencing to help bring an infectious disease outbreak in a hospital to a close. Researchers used advanced DNA sequencing technologies to confirm the presence of an ongoing outbreak of methicillin-resistant Staphylococcus aureus (MRSA) in a Special Care Baby Unit in real time. This assisted in stopping the outbreak earlier, saving possible harm to patients. This approach is much more accurate than current methods used to detect hospital outbreaks.Tue, 13 Nov 2012 21:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121113214635.htmhttp://www.sciencedaily.com/releases/2012/11/121113161506.htm Findings from a study suggest that certain variations in vitamin D metabolism genes may modify the association of low serum 25-hydroxyvitamin D concentrations with health outcomes such as hip fracture, heart attack, cancer, and death.Tue, 13 Nov 2012 16:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121113161506.htmhttp://www.sciencedaily.com/releases/2012/11/121113143656.htm Biologists have discovered that fats within cells store a class of proteins with potent antibacterial activity, revealing a previously unknown type of immune system response that targets and kills bacterial infections.Tue, 13 Nov 2012 14:36:36 ESThttp://www.sciencedaily.com/releases/2012/11/121113143656.htmhttp://www.sciencedaily.com/releases/2012/11/121113134807.htm Researchers using a new approach to identifying genes associated with depression have found that variants in a group of genes involved in transmission of signals by the neurotransmitter glutamate appear to increase the risk of depression.Tue, 13 Nov 2012 13:48:48 ESThttp://www.sciencedaily.com/releases/2012/11/121113134807.htmhttp://www.sciencedaily.com/releases/2012/11/121113134230.htm The cancer-causing form of the gene Myc alters the metabolism of mitochondria, the cell?s powerhouse, making it dependent on the amino acid glutamine for survival. Depriving cells of glutamine selectively induces programmed cell death in cells overexpressing mutant Myc. Using Myc-active neuroblastoma cells, a team three priotein executors of the glutamine-starved cell, representing a downstream target at which to aim drugs. Roughly 25 percent of all neuroblastoma cases are associated with Myc-active cells.Tue, 13 Nov 2012 13:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121113134230.htmhttp://www.sciencedaily.com/releases/2012/11/121113134224.htm Even the very lowest levels of radiation are harmful to life, scientists have concluded, reporting the results of a wide-ranging analysis of 46 peer-reviewed studies published over the past 40 years. Variation in low-level, natural background radiation was found to have small, but highly statistically significant, negative effects on DNA as well as several measures of health.Tue, 13 Nov 2012 13:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121113134224.htm

Source: http://www.sciencedaily.com/rss/health_medicine/genes.xml

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